Jackob Moskovitz

Jackob Moskovitz

Mar 19, 2019

Group 6 Copy 151
0
    Please wait...

    About This Project

    Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

    Blast off!

    Browse Other Projects on Experiment

    Related Projects

    Are climate change and air pollution triggering cardiovascular disease?

    Ongoing global climate change and air pollution emissions pose a major threat to cardiovascular health...

    Developing a new tissue preservation method inspired by Tardigrades

    There are two routine ways to preserve tissues: formalin-fixed paraffin-embedded preservation (FFPE) and...

    Predicting and improving persistence during exposure-based therapy for OCD

    The efficacy of exposure-based therapies for obsessive compulsive disorder (OCD) is among the most well...

    Campaign Ended

    A medicine project funded by 45 people