Jackob Moskovitz

Jackob Moskovitz

Apr 11, 2019

Group 6 Copy 264
-1
    Please wait...

    About This Project

    Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

    More Lab Notes From This Project

    Blast off!

    Browse Other Projects on Experiment

    Related Projects

    Developing a low-cost, rapid diagnostic for urogenital Schistosomiasis infection

    We are developing a frugal diagnostic for urogenital schistosomiasis, a neglected tropical disease that...

    Understanding immune system paralysis in times of bacterial stress

    Sepsis is a life-threatening condition in which the body's immune response to infection injures its own...

    How does taking Ritalin affect depersonalization-derealization disorder patients?

    We are investigating Ritalin's role in alleviating symptoms of depersonalization-derealization (DPDR) disorder...

    Campaign Ended

    A medicine project funded by 45 people