Jackob Moskovitz

Jackob Moskovitz

Mar 06, 2019

Group 6 Copy 191
1
Please wait...

About This Project

Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

More Lab Notes From This Project

Blast off!

Browse Other Projects on Experiment

Related Projects

How does taking Ritalin affect depersonalization-derealization disorder patients?

We are investigating Ritalin's role in alleviating symptoms of depersonalization-derealization (DPDR) disorder...

Early-Stage Drug Discovery for Chagas' Disease

An estimated 6 – 7 million people in Latin America and 300,000 U.S. citizens are infected with the protozoan...

Will vitamin D improve the health of patients with heart failure?

We are trying to determine if supplemental vitamin D in heart failure patients will improve heart function...

Campaign Ended

A medicine project funded by 45 people