Jackob Moskovitz

Jackob Moskovitz

Apr 23, 2019

Group 6 Copy 131
1
Please wait...

About This Project

Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

Blast off!

Browse Other Projects on Experiment

Related Projects

Antibiotic Resistance: Between the Environment and Our Bodies

Bacteria have evolved many genes to make them resistant to most of our antibiotics. Such genes can be moved...

Can a Dietary Intervention Reduce Mercury Toxicity Among Native Communities in Peru?

A state of emergency has been declared in Madre de Dios, Peru due to high levels of mercury (Hg) exposure as...

From Minerals to Medicines: A New Technology for Heavy Metal Pollution Remediation

This is the first part of a multi-phase experiment in which I will prove that substances isolated from plant...

Campaign Ended

A medicine project funded by 45 people