Please wait...
About This Project
Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.
More Lab Notes From This Project

Browse Other Projects on Experiment
Related Projects
Developing a novel oxysterol antibiotic to combat drug-resistant tuberculosis
Drug-resistant tuberculosis (TB) is a consistently growing threat to global health. We have developed Oxy291...
Developing a low-cost, rapid diagnostic for urogenital Schistosomiasis infection
We are developing a frugal diagnostic for urogenital schistosomiasis, a neglected tropical disease that...
Making Inexpensive Insulin Using Microalgae
This page serves as an archive of our first proof-of-concept experiments to genetically modify microalgae...