Jackob Moskovitz

Jackob Moskovitz

Mar 21, 2019

Group 6 Copy 174
-1
    Please wait...

    About This Project

    Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

    More Lab Notes From This Project

    Blast off!

    Browse Other Projects on Experiment

    Related Projects

    Can personalized medicine subtype bladder cancer?

    Molecular pathoepidemiology is the study of the cause and natural history of disease on a molecular, individual...

    Can a rabies vaccination booster save African Wild Dogs?

    Rabies is one of the greatest threats to the continued survival of the African wild dog (Lycaon pictus...

    Can cockroaches help us cure tularemia?

    Tularemia is a deadly disease caused by the bacterium Francisella tularensis (Ft). This bacterium is also...

    Campaign Ended

    A medicine project funded by 45 people