Jackob Moskovitz

Jackob Moskovitz

Mar 06, 2019

Group 6 Copy 166
1
Please wait...

About This Project

Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

Blast off!

Browse Other Projects on Experiment

Related Projects

Can a Dietary Intervention Reduce Mercury Toxicity Among Native Communities in Peru?

A state of emergency has been declared in Madre de Dios, Peru due to high levels of mercury (Hg) exposure...

Next Generation Non-Seeing Eye Prosthesis

This project aims to push the current state of the art in ocular prothestics by integrating a small system...

S-glutathionylation reactions in mitochondrial function and disease

This project was an invited review that is currently under minor revision at Frontiers Cellular and Developmental...

Campaign Ended

A medicine project funded by 45 people