Jackob Moskovitz

Jackob Moskovitz

Mar 06, 2019

Group 6 Copy 165
1
Please wait...

About This Project

Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.

Blast off!

Browse Other Projects on Experiment

Related Projects

A Prescription for Health and Fitness Based on Your Genes

Exercise has many health benefits. Yet, most people stop their exercise programs after they start them. What...

Help us pioneer stem cell therapy for giraffe!

Arthritis is common in geriatric giraffe. Cheyenne Mountain Zoo is uniquely suited to help find better arthritis...

Can we predict whether an athlete will sustain an ACL tear?

Despite consistent investigation, the risk factors responsible for non-contact Anterior Cruciate Ligament (ACL...

Campaign Ended

A medicine project funded by 45 people