Kylee Tucholski

Kylee Tucholski

Oct 15, 2019

Group 6 Copy 14
0
    Please wait...

    About This Project

    The hypothesis that is being tested is if C. elegans will build up amyloid beta (AB) plaques after heat stress exposure, possibly leading to Alzheimer's disease (AD). C. elegans (nematodes) demonstrate symptoms of Alzheimer’s disease such as amyloid plaques, genes and their mutations, and memory impairments. Heat-shock is a form of stress that could potentially increase the rate of AD, and during heat shock, transcription and translation proteins in C. elegans will become inhibited.

    Recent Lab Notes From This Project

    Blast off!

    Browse Other Projects on Experiment

    Related Projects

    How does the brain quickly recognize threat and can we improve it?

    Understanding and responding to threatening emotions is important in a wide range of situations faced by humans...

    Identifying the genes necessary to regenerate an injured brain - insights from a basal chordate, Ciona intestinalis

    Only a few regions of the mammalian brain are capable of replacement by neural stem cells, therefore it is important...

    Amelioration of Alpha-Synuclein in Parkinson's Disease through potentiated protein-protein interactions

    Aggregation of misfolded alpha synuclein protein is a key feature of Parkinson's Disease (PD), which afflicts...